Liver Aid


Tauroursodeoxycholic Acid-TUDCA is a water soluble bile acid. It shows great potency in treating cholestasis (bile acid backup in the liver) as the water soluble bile acids counteract the toxicity of regular bile acids. Can also protect and rehabilitate the liver, and general protects cells. Itís used as a therapy for people that have any type of issue with the liver or liver bile duct. You only have small amounts of this amazing compound in your body.

Hell's Burn

Increasing the amount of TUDCA in your body can help clear up issues you are having with your liver.When the liver is under high stress because of a blocked bile duct, your liver enzymes can elevate significantly. Another TUDCA benefit is lowering these liver enzymes. One study showed that even a modest TUDCA dose can dramatically lower your liver enzymes in two months. TUDCA helps increase thyroid function. It helps upregulate energy in the cells. Brown adipose tissue is more metabolically active than white. Study showed that TUDCA can double the amount of brown adipose tissue. TUDCA increases the amount of energy the body can use up and helps to use insulin better. TUDCA helps the heart.High levels of sugar in the blood because of poor insulin usage doesnít just affect body weight. High glucose levels damage endothelial cells. These are the cells that line the inside of all body organs and blood vessels. This damage increases the chance for cardiovascular disease.

TUDCA helps protect these special cells that line heart and arteries. It reduces oxidative stress. It prevents the endothelial cells from becoming dysfunctional in the presence of too much glucose.


The active ingredients in milk thistle are a group of plant compounds collectively known as silymarin. Its herbal remedy is known as milk thistle extract. Milk thistle extract has a high amount of silymarin (between 65Ė80%) that has been concentrated from the milk thistle plant. Milk thistle is often promoted for its liver-protecting effects. Itís regularly used as a complementary therapy by people who have liver damage due to conditions like alcoholic liver disease, non-alcoholic fatty liver disease, hepatitis and even liver cancer. Itís also used to protect the liver against toxins like amatoxin, which is produced by the death cap mushroom and is deadly if ingested.


L-ornithine-L-aspartate increases levels the ornithine and aspartic acid in the body. These amino acids help to reduce levels of a toxic chemical called ammonia in the blood. L-ornithine- L-aspartate (LOLA), the salt of the natural amino acids ornithine and aspartate acts through the mechanism of substrate activation to detoxify ammonia. In clinical trials, LOLA has shown a statistically significant effect with respect to reduction in HE (Hepatic encephalopathy) grade, reduction of blood ammonia concentration and positive effects on psychomotor function in patients of cirrhosis with minimal HE and overt chronic Grade I HE.

4. NAC (N-acetylcysteine)

Is a modified amino acid that is used to reverse the toxic effects of acetaminophen overdose and prevent acute liver failure. The amino acid cysteine has a thiol side chain that can undergo redox reactions and thus has antioxidant activity.Cysteine is required for synthesis of glutathione (a tripeptide of cysteine, glycine and glutamic acid) that is an essential intracellular antioxidant, providing protection against free radicals and other intracellular toxins including intermediates of drug metabolism. Glutathione can become depleted in patients with malnutrition, chronic debilitating illnesses and chronic alcoholism, and becomes acutely depleted in patients with acetaminophen overdose leading to formation of toxic adducts of acetaminophen metabolites with essential intracellular molecules. Restoration of glutathione stores ameliorates the cell injury from acetaminophen.Administration of acetylcysteine (also referred to as N-acetylcysteine or NAC) within 10 hours of an acetaminophen overdose effectively prevented serious liver injury. Subsequent studies suggested that administration even after 10 hours was partially effective and may also be beneficial in other, non-acetaminophen causes of acute liver failure, including drug induced liver injury.